Best Varicose Veins Treatment in Jaipur
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Varicose veins are enlarged veins near the skin’s surface, often found in the legs due to increased pressure from standing and walking. For Best Varicose Veins Treatment in Jaipur, Dr. Manish Rajput provides advanced, personalized care using modern techniques to help you achieve healthier, smoother legs and a better quality of life.
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✅ MVP completed
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We are currently raising $5M to scale across the U.S., EU, IL and MENA.
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Introduction
Lipid autocoids, also known as eicosanoids and related lipid mediators, are bioactive molecules derived from polyunsaturated fatty acids (PUFAs). These molecules play crucial roles in inflammation, immunity, hemostasis, cardiovascular function, and various physiological and pathological processes. Unlike classical hormones, lipid autocoids act locally, exerting their effects at or near their site of synthesis.
This document provides an in-depth analysis of lipid autocoids, covering their biosynthesis, classification, physiological roles, and clinical significance.
Classification of Lipid Autocoids
Lipid autocoids are broadly classified into the following categories:
1. Eicosanoids (Derived from Arachidonic Acid)
Prostaglandins (PGs)
Thromboxanes (TXs)
Leukotrienes (LTs)
Lipoxins (LXs)
2. Specialized Pro-resolving Mediators (SPMs)
Resolvins
Protectins
Maresins
3. Endocannabinoids
Anandamide (AEA)
2-Arachidonoylglycerol (2-AG)
4. Platelet-Activating Factor (PAF)
5. Sphingolipid-Derived Mediators
Sphingosine-1-Phosphate (S1P)
Ceramides
Biosynthesis of Lipid Autocoids
Lipid autocoids are derived from membrane phospholipids through enzymatic pathways:
1. Phospholipase A2 (PLA2) Activation:
PLA2 catalyzes the release of arachidonic acid (AA) from membrane phospholipids.
2. Cyclooxygenase (COX) Pathway:
Converts AA into prostaglandins and thromboxanes.
COX-1: Constitutive enzyme (housekeeping functions).
COX-2: Inducible enzyme (inflammation and pain response).
3. Lipoxygenase (LOX) Pathway:
Converts AA into leukotrienes and lipoxins.
5-LOX: Leads to leukotrienes (inflammation, bronchoconstriction).
12-LOX & 15-LOX: Lead to lipoxins (anti-inflammatory action).
4. Cytochrome P450 (CYP) Pathway:
Converts AA into epoxyeicosatrienoic acids (EETs), which regulate vascular tone.
5. Endocannabinoid Biosynthesis:
Derived from membrane phospholipids via enzymatic reactions.
Degraded by fatty acid amide hydrolase (FAAH) and monoacylglycerol lipase (MAGL).
Physiological Roles of Lipid Autocoids
1. Inflammation and Immune Response
Prostaglandins (e.g., PGE2) modulate fever and pain.
Leukotrienes mediate allergic responses and asthma.
Lipoxins and resolvins promote resolution of inflammation.
2. Cardiovascular System
Thromboxanes (TXA2) induce platelet aggregation and vasoconstriction.
Prostacyclin (PGI2) inhibits platelet aggregation and promotes vasodilation.
EETs regulate blood pressure and vascular homeostasis.
3. Pulmonary Function
Leukotrienes (LTC4, LTD4, LTE4) are potent bronchoconstrictors.
PGE2 has bronchodilatory effects.
4. Renal Function
Prostaglandins regulate glomerular filtration rate and sodium excretion.
EETs contribute to natriuresis.
5. Neurotransmission and Pain
Endocannabinoids modulate pain perception and neuroprotection.
Prostaglandins contribute to central pain sensitization.
6. Reproductive System
P
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Pharmacoeconomics is a vital field that examines the economic impact of pharmaceutical products and healthcare services. This presentation provides a detailed overview of pharmacoeconomic principles, methodologies, and their significance in healthcare decision-making. It covers essential topics such as cost analysis, evaluation perspectives, and humanistic assessment methods.
💡 Key Topics Covered:
✔ History and Evolution of Pharmacoeconomics
✔ Goals and Objectives of Pharmacoeconomic Studies
✔ Cost Analysis & Consequences (Outcomes)
✔ Different Pharmacoeconomic Methodologies (Cost-Minimization, Cost-Effectiveness, Cost-Utility, Cost-Benefit)
✔ Perspectives in Economic Evaluations (Payer, Patient, Society)
✔ Role of Pharmacoeconomics in Drug Safety & Pharmacovigilance
✔ Humanistic Evaluation Methods (Quality of Life & Patient-Reported Outcomes)
✔ Importance of Pharmacoeconomics in Policy & Healthcare Decisions
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Rheumatology: Understanding Rheumatoid Arthritis & Physiotherapy ManagementEndorphaminee his presentation provides an in-depth look into Rheumatoid Arthritis (RA), a chronic autoimmune condition that primarily affects the joints, causing pain, stiffness, and potential deformities. It explores the pathophysiology, diagnosis, and symptoms of RA, providing a clear understanding of how the disease progresses and the impact it has on patients' lives.
Additionally, this presentation focuses on the role of physiotherapy in managing RA. It covers various physiotherapy techniques, including exercise therapy, manual therapy, and modalities such as heat/cold therapy and TENS, which help improve joint mobility, reduce pain, and enhance overall function. It also emphasizes the importance of patient education and the need for a multidisciplinary approach to effectively manage RA.
This resource is designed for healthcare professionals, students, and individuals interested in understanding both the medical and rehabilitation aspects of Rheumatoid Arthritis. Whether you’re looking to enhance your knowledge of RA or seeking effective physiotherapy strategies for management, this presentation serves as a comprehensive guide.
Drug_Design PRESENTSTION B.pharm 6th sem. IGNTU AMARKANTAK M.P.
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SYNAPSE AND NEUROTRANSMITTERS (Nervous System).pptxPranaliChandurkar2 There is always more than one neurone involved in the transmission of a nerve impulse from its origin to its destination, whether it is sensory or motor.
There is no physical contact between two neurones.
Synapse: The point at which the nerve impulse passes from the presynaptic neurone to the postsynaptic neurone.
Presynaptic Neurons: The neurons that carries action potential to the synapse.
Postsynaptic Neurons: Neurons that carries action potential away from the synapse.
Synaptic knobs or terminal boutons: At its free end, the axon of the presynaptic neurone breaks up into minute branches that terminate in small swellings.
These are in close proximity to the dendrites and the cell body of the postsynaptic neurone.
The space between them is the synaptic cleft.
Synaptic knobs contain spherical membrane bound synaptic vesicles, which store a chemical (the neurotransmitter) that is released into the synaptic cleft.
Neurotransmitters are synthesised by nerve cell bodies, actively transported along the axons and stored in the synaptic vesicles.
They are released by exocytosis in response to the action potential in the presynaptic neurons and diffuse across the synaptic cleft.
They act on specific receptor sites on the postsynaptic membrane.
Their action is short lived, because immediately they have acted on the postsynaptic cell such as a muscle fibre, they are either inactivated by enzymes or taken back into the synaptic knob.
Some important drugs act by mimicking, neutralising (antagonising), or prolonging normal actions of neurotransmitters.
Neurotransmitters usually have an excitatory effect on postsynaptic receptors but they are sometimes inhibitory.
There are more than 50 neurotransmitters in the brain and spinal cord including noradrenaline (norepinephrine), adrenaline (epinephrine), dopamine, histamine, serotonin, gamma aminobutyric acid (GABA) and acetylcholine.
Other substances, such as Neuropeptides (enkephalins, endorphins and substance P) have specialised roles in, for example, transmission of pain signals.
Excitatory neurotransmitters: Acetylcholine, Norepinephrine, Dopamine (Initiate New Impulse)
Inhibitory neurotransmitters: GABA, Serotonin, Glycine (Reduces the chance of new impulse, Hyperpolarization).
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Best Varicose Veins Treatment in Jaipur
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